Updating the natural history of hpv and anogenital cancer dating is horrible

Although these studies have demonstrated the importance of HPV persistence in the development of precancer CIN-3, the timing from infection to evidence of CIN-3 varies from 1 to 10 years.

updating the natural history of hpv and anogenital cancer-23

Enzyme-linked immunosorbent assays (ELISAs) utilising virus-like particles have been used successfully for seroprevalence studies in several countries including the USA (Stone et al, 2002) and Sweden (af Geijersstam et al, 1999).

We report on the first population-based study of HPV 6, 11, 16 and 18 seroprevalence in England, in 10- to 29-year-old female subjects – the likely target age range for vaccination, but an age range in which little is known about infection rates.

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This chapter addresses the natural history of anogenital human papillomavirus (HPV) infection.

Cervical infections are the best understood HPV infection.

This means that essentially all cervical cancers are associated with persistent infection with high-risk HPV.

High-risk types are associated with cervical and other cancers, whereas low-risk types are associated with external genital warts but not cancer.

These HPV types (see Table 1) have been shown to be carcinogenic for the cervix.

As shown in Figure 1, HPV prevalence varies by age and is highest for young women, decreasing in the middle age range (30 to 60 years old).

The importance of the transformation zone in cervical cancer has been extended to other HPV-induced cancers such as anal or tonsillar cancers.

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